As crianças com baixa estatura familiar têm pais baixos. Estas crianças são normais e exibem a velocidade de crescimento normal (velocidade de crescimento ao longo do tempo), e o seu desenvolvimento ósseo é normal (tal como indicado pela idade óssea correspondente à idade cronológica). As crianças com baixa estatura familiar entram na puberdade em um tempo normal e têm o crescimento tipicamente completo com uma estatura compatível com a de seus pais, mas também não podemos nos esquecer de avaliar seus tios, primos, avós e seus outros ancestrais.
THE CAUSES OF THE COMMITMENT Of LINEAR AND LONGITUDINAL GROWTH IN CHILDREN, CHILD AND YOUTH.
THE LOW HEIGHT IN SHORT TERM (EARLY) DESCRIBES THE HEIGHT THAT IS SIGNIFICANTLY BELOW AVERAGE THE HEIGHT FOR AGE OF A PERSON, GENDER, RACIAL GROUP OR FAMILY AT ANY STAGE OF GROWTH OF CHILD, CHILDREN, JUVENILE OR TEENAGER:. PHYSIOLOGY-ENDOCRINOLOGY-NEUROENDOCRINOLOGY-GENETICS-ENDOCRINE-PEDIATRICS (SUBDIVISION OF ENDOCRINOLOGY): DR. JOÃO SANTOS CAIO JR. ET DRA. HENRIQUETA VERLANGIERI CAIO.
Prior research suggests that children who suffer from multiple transitions in family structure may face worse outcomes of development than children raises in stable families with both parents and brothers, perhaps even raised in stable, single-parent families. However, a number of transitions and negative results could be associated with children because of common causative factors, such as behaviors and attributes of the parent record. Using a two generation study nationally representative, longitudinal, which includes detailed information on the behavioral and cognitive development of the child, family history, and the attributes of the mother before the child's birth, we examine these alternative hypotheses.
The lack of growth is often confused with short stature. The lack of growth that occurs over time, possibly resulting in short stature. By definition, the growth failure is a medical condition. However, the short stature is often a normal variation but should be investigated. The short stature may or may not be a sign or symptom of a medical condition caused. Evaluation of growth over time, not just at a single point in time is essential in maintaining the health of children. Any deviation from a pattern of growth earlier genetically suitable for the child can signal the onset of a disease. All causes of the short stature, with a medical condition, fall into one of three main categories: chronic disease (e.g., malnutrition), familial short stature or constitutional delay of growth and development ("Late Bloomers"). Worldwide, malnutrition is the most common cause of failure to growth and is usually related to poverty. Nutritional deficiencies in developing countries are more often the result of self restricted diets. Weight gain is often more noticeable than the short stature. Children with familial short stature have short parents. These normal children exhibit normal growth rate (growth rate over time), and their bone development is normal (as indicated by bone age corresponding to chronological age). The children with familial short stature enter puberty at a normal time and are typically full growth with a compatible stature of their parents, but we cannot forget to evaluate your uncles, cousins, grandparents and his other ancestors.
Dr. João Santos Caio Jr.
Endocrinologia – Neuroendocrinologista
CRM 20611
Dra. Henriqueta V. Caio
Endocrinologista – Medicina Interna
CRM 28930
1. A causa de baixa estatura longitudinal ou linear pode ser deficiência de vitamina D devido à ingestão oral inadequada, má absorção de gordura, exposição solar inadequada, terapia anticonvulsivante e/ou doenças hepáticas ou renais...
http://hormoniocrescimentoadultos.blogspot.com
2. Os achados clássicos de raquitismo com deficiência de vitamina D incluem encurvamento das pernas, deformidades torácicas (rosário raquítico) e achados radiográficos característicos das extremidades associados à redução do cálcio sérico e de fosfato e elevação dos níveis séricos de fosfatase alcalina...
http://longevidadefutura.blogspot.com
3. Existem 2 formas hereditárias dependentes de Vitamina D: o raquitismo hipofosfatêmico autossômico recessivo tipo 1 (OMIM # 241520) que envolve uma deficiência de 25 OHD1-hidroxilase. E o tipo 2 (OMIM # 613312) que envolve um receptor de vitamina D deficiente ou ausente...
http://imcobesidade.blogspot.com
AUTORIZADO O USO DOS DIREITOS AUTORAIS COM CITAÇÃO
DOS AUTORES PROSPECTIVOS ET REFERÊNCIA BIBLIOGRÁFICA.
Referências Bibliográficas:
Caio Jr, João Santos, Dr.; Endocrinologista, Neuroendocrinologista, Caio,H. V., Dra. Endocrinologista, Medicina Interna – Van Der Häägen Brazil, São Paulo, Brasil; Swanson HL, Malone S. Social skills and learning disabilities: A meta-analysis of the literature. School Psych Rev. 1992;21:427–441; Lopez MF, Forness SR. Children with attention deficit hyperactivity disorder or behavioral disorders in primary grades: Inappropriate placement in the learning disability category. Ed Treat Child. 1996;19:286–299; Geary DC. Mathematical disabilities: Cognitive, neuropsychological, and genetic components. Psych Bull. 1993;114:345–362; Geary DC. Mathematics and learning disabilities. J Learn Dis. 2004;37:4–15; Jordan NC, Montani TO. Cognitive arithmetic and problem solving: A comparison of children with specific and general mathematics difficulties. J Learn Dis. 1997;30:624–634; Butterworth B. Developmental dyscalculia. In: Campbell JID, editor. Handbook of mathematical cognition. New York: Psychology Press; 2005; Rourke BP. Arithmetic disabilities, specific and otherwise: A neuropsychological perspective. J Learn Dis. 1993;26:214–226; Temple CM. Procedural dyscalculia and number fact dyscalculia: Double dissociation in developmental dyscalculia. Cog Neuropsych. 1991;8:155–176; Geary DC. Missouri longitudinal study of mathematical development and disability. Brit J Ed Psych Mono Ser II. 2010;7:31–49; Geary DC, Hoard MK, Nugent L, Bailey DH. Mathematical cognition deficits in children with learning and intellectual disabilities: A five year prospective study. Under editorial review; Geary DC. Reflections of evolution and culture in children’s cognition: Implications for mathematical development and instruction. Am Psych. 1995;50:24–37.
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